Scientists from the Korean Institute of Advanced Technologies (Kaist) created a protein that intervened in the restoration of the retina’s nerve cells and creates a drug that could prevent the effect. Research Results published In Nature Communications (Natcom).
Glaucoma affects millions of people, including the elderly, such as age -related macular degeneration and diabetic retinopathy. One of the promising areas of ophthalmology is the restoration of its own eye cells. However, so far, researchers have not overcome natural restrictions on retinal regeneration.
Scientists have managed to determine that the key barrier is a prox1 protein that prevents the regenerative activity of mullet cells – a special gial cell in the retina. These cells are known for their ability to participate and participate in the protection and protection of eye structure.
A new approach uses a drug based on antibodies that suppress the activity of prox1. In experiments on laboratory rats, protein blocking made it possible to activate the restoration of damaged retinal tissues. The effect was preserved for at least six months.
Development has become a part of the global tendency in medicine – the body’s search for ways to activate its own restoration mechanisms. Unlike methods based on cell transplantation or laser therapy, this approach allows you to initiate “inside” regeneration, which makes it potentially safer and more universal.
Previously, scientists learnedBlind people can “see” colors through language.
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Source: Gazeta
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