Researchers from California State University in the USA discovered that neurons in the nucleus tractus solitarius in the brain can control feeding behavior in mice. results published in nature.
Scientists tried to find out what exactly is the mechanism that informs the brain about fullness and signals the need to stop eating. To do this, they conducted an experiment using mice. After a series of experiments, scientists found that only two types of neurons located in the nucleus of the solitary system are involved in natural appetite suppression. These are GCG neurons, which “control” the amount of food consumed, and PRLH, which “fixes” the processes of chewing and swallowing.
Neurons of the first type are activated by signals from the intestines as the stomach fills with food masses. They are the ones who report that saturation has been reached. PRLH “works” as a stimulant when food enters the oral cavity. They become most active when consuming sweet, fatty and high-calorie foods. However, unlike GCG, PRLH neurons do not influence food intake. Its activities are aimed at regulating the rate of eating, which helps prevent gastrointestinal disorders.
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